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Cermakian and his colleagues looked for the current presence of the circadian time clock genes, PER1, PER2, and BMAL1 in the brain cells of 58 donors; 27 were Alzheimer individuals and 31 were settings. All these clock genes had been detected in three mind areas in both Alzheimer and control patients. Furthermore, there was a substantial diurnal expression of the genes in every three structures of control samples. In samples from Alzheimer’s patients, the shape of these rhythms and the synchronization between brain areas were altered. ‘The abnormal clock gene coordination that we observed in the tissues of Alzheimer’s patients, might describe the sleep-wake deficits that are observed in this population,’ says Douglas Centre for Treatment and Research of Circadian Rhythms Director, Diane B.